----- Begin Included Message -----
7. British-Hungarian Science & Technology Programme 1996-1999
- British Council
The British-Hungarian Intergovernmental Programme of
Science & Technology will be renewed in March 1996. It is
expected to agree to fund collaborative research projects.
Priority will be given to projects in the following areas;
environmental protection, biotechnology, agricultural
sciences, energy, information technology, medical science,
science & technology policy, and technology
diffusion/innovation. Awards will be of 2-3 years duration
and will cover only travel & subsistence costs relating to
the project.
Parallel applications should be submitted by the UK partner
to the British Council in Hungary, and by the Hungarian
partner to the Hungarian Committee for Technological
Development (OMFB).
Application forms are available from:
The British Council
Budapest VI
Benczur utca 26
H-1068 Hungary
Tel: 00 36 1 321 4039
Fax: 00 36 1 342 5728/269 6594
Closing Date: 23 November 1995
----- End Included Message -----
Luca Lorenzo Bonatti
of the cognitive science group at the CNRS/Ecole Pratique in
Paris is giving an intsensive seminar as part of our Cognitive
Doctoral program.
TOPIC: New Models of Human Reasoning
Time: from 11th to 23rd of September.
Daily seminars at an hour to be specified later.
Luca will be `stationed` in Izabella street.
Details of the work will be specified later. This preliminary
anouncement is to let us know two things:
- who would like to ettend.
- what our would be preferable.
Please notify both luca and pleh at the email adresses below.
Please post this note.
For cognitive psychology majors the class is a must.
Tentative listing:
THE PSYCHOLOGY OF REASONING
1. Introduction.
2. Options for a theory of reasoning: structural and non
structural approaches. (Core references: Rips 1990; McGinn 1989;
Rumelhart 1989;Bechtel & Abrahansen 1992, ch. on reasoning)
3. Theories of propositional reasoning. A. Mental logic: the work
of Rips and Braine (Core references: Rips 1994, ch. 4 and 5;
Marcus (1982) Braine. Reiser & Rumain 1984; Braine & O'Brien
forthcoming; Braine,O'Brien, Noveck, Samuels, Fish,& Yang,
forthcoming; Brooke Lea et al. 1990)
4. Theories of propositional reasoning. B. Mental models (Core
references: Johnson-Laird, Byrne & Schaeken (1991, chs. 3, 4;
1992; 1994);Bauer & Johnson-Laird 1993)
5. Theories of propositional reasoning. C. A connectionist model:
SHRUTI. Conclusion on propositional reasoning: pure and impure
representations. (Core references: Shastri & Ajjanagadde 1994,
and related discussion).
6. Figural effects and syllogistic reasoning (Core references:
Johnson-Laird & Bara 1984; Ford 1995; Rips 1994; Newstead 1993)
7. The Belief Bias effect and the suppression of valid inferences
effect (Core references: Oakill & Johnson-Laird 1985; Newstead,
Pollard, Evans, & Allen,1992; Evans, Barston & Pollard 1983;
Newstead & Evans 1992; Byrne 1989; 1991)
8. Reasoning and the selection task. (Core references:
Johnson-Laird & Legrenzi 1972; Wason 1983; Griggs & Newstead
1983; Griggs 1983; Cheng & Holyoak 1985; Cosmides 1989;
Gigerenzer & Hug 1992; Johnson-Laird and Byrne
1991, ch. 4; Sperber, Cara & Girotto, Forthcoming; Rips 1994, ch.
4 )
9. Between deductive reasoning and task-specific reasoning: the
case of spatial reasoning. (Core references: Franklin & Tversky
1990; Taylor & Tversky 1992; Bryant, Tversky& Lanca, MS )
10. Developmental and neurological evidence for reasoning: the
state of the art (Core references: Bowerman 1986; Braine & Rumain
1983; neurological literature)
pleh(a)izabell.elte.hu
luca(a)msh-paris.fr
====================
Luca L. Bonatti
LSCP-MSH
54, Bd. Raspail
75006 Paris
France
off. #: (33) (1) 4954-2068
ho. #: (33) (1) 4263-0149
====================
>
>> Coming in April 1996
>> THE TENTH BIENNIAL INTERNATIONAL CONFERENCE ON INFANT STUDIES
>> Program Chair: J. Steven Reznick
>>
>> April 18,-21, 1996
>> WESTIN HOTEL
>> Providence, Rhode Island
>>
>> For more information and/or to be placed on the mailing list,
>>
>> contact: Lewis P. Lipsitt, Ph.D.
>> 1996 ICIS Conference Chair
>> Brown University, Box 1853
>> Providence, RI 02912 USA
>>
>> Telephone: (401)863-2332
>> FAX: (401)863-1300
>>
>> e-mail: LLIPSITT(a)BROWNVM.BROWN.EDU
>>
>
Kedves Kollegak,
igen surgosen szeptembertol lenne lehetoseg egy gyakornoki allasra.
Feltetelek a kovetkezok:
(1) Pszichologus vegzettseg.
(2) Nyelvtudas.
(3) Kiserleti pszichologiai orientacio, s nemi matematikai charme.
Kerem, az erdeklodok minel hamarabb keressenek:
pleh(a)izabell.elte.hu
illetve Izabella utca 46, III. emelet 316.
Peldaul hetfon 28 vagy kedden 29-en.
Az allas nem az ELTEn van, magam csak szakmai kozvetito vagyok a
dologban.
Kedves Csaba!
Orommel olvastam az allashirdetest. Ez azt jelenti, hogy Englander
Tibor megfogadta a tanacsot es Hozzad fordult. Remelem tudsz segiteni
neki.
Udvozlettel: Gyurka
--
*****************************************************************
George Karmos M.D., Ph.D. Mail: P.O.Box 398,
Budapest
Institute for Psychology of the H-1394 Hungary
Hungarian Academy of Sciences Phone: +36-1 153-3244
Fax: +36-1 269-2972
Szondi u. 83-85. e-mail: karmos(a)cogpsyphy.hu
Budapest VI., Hungary
*****************************************************************
I have an apartment for longterm (at least 1 year) sublet.
1 bedroom, telefon, washer, spectacular views on the Danube and
Szt Istvan park. Telefon: 1495-081.
Balog Katalin
Pesten vagyok szept. 1-ig!
Below are the abstracts for 8 forthcoming target articles on
MOTOR LEARNING AND SYNAPTIC PLASTICITY
to appear in BBS's Special Issue: Controversies in Neurosciences IV
[commentators may comment on one, several, or all 8 target articles]
(1) bbs.crepel: CELLULAR MECHANISMS OF LONG-TERM DEPRESSION IN THE CEREBELLUM
(2) bbs.houk: MODELS OF THE CEREBELLUM AND MOTOR LEARNING
(3) bbs.kano: POTENTIATION OF GABAERGIC INHIBITORY TRANSMISSION PURKINJE CELLS
(4) bbs.linden: CEREBELLAR LONG-TERM DEPRESSION IN CELL CULTURE
(5) bbs.simpson: ON CLIMBING FIBER SIGNALS AND THEIR CONSEQUENCE(S)
(6) bbs.smith: CEREBELLAR LEARNING AND OPTIMAL JOINT STIFFNESS CONTROL
(7) bbs.hach: CEREBELLUM, MOTOR LEARNING AND THINKING IN MAN: PET STUDIES
(8) bbs.vincent: NITRIC OXIDE AND SYNAPTIC PLASTICITY: NO NEWS FROM CEREBELLUM
These 8 articles have been accepted for co-publication in Behavioral
and Brain Sciences (BBS), an international, interdisciplinary journal
providing Open Peer Commentary on important and controversial current
research in the biobehavioral and cognitive sciences.
Commentators must be current BBS Associates or nominated by a current
BBS Associate. To be considered as a commentator for this article, to
suggest other appropriate commentators, or for information about how to
become a BBS Associate, please send email to:
bbs(a)ecs.soton.ac.uk or write to:
Behavioral and Brain Sciences
Department of Psychology
University of Southampton
Highfield, Southampton
SO17 1BJ UNITED KINGDOM
http://cogsci.ecs.soton.ac.uk/~harnad/bbs.html
gopher://gopher.princeton.edu:70/11/.libraries/.pujournals
ftp://ftp.princeton.edu/pub/harnad/BBS
To help us put together a balanced list of commentators, please give
some indication of the aspects of the topic on which you would bring
your areas of expertise to bear if you were selected as a commentator.
Electronic draft of the full texts of all 8 are available for inspection by
anonymous ftp (or gopher or [best method] WorldWide Web) according to the
instructions that follow after the 8 abstracts.
Commentators may comment on one, several, or all 8 target articles.
Commentary length is 1000 words for a commentary on one target article,
to a maximum of 2000 words for a commentary on all 8 (i.e. about 140
additional words for each additional target article).
(1) CELLULAR MECHANISMS OF LONG-TERM DEPRESSION IN THE CEREBELLUM
F. Crepel, N. Hemart, D. Jaillard and H. Daniel
Laboratoire de Neurobiologie et
Neuropharmacologie du Dveloppement,
CNRS URA 1121, Bat. 440,
Universit Paris-Sud,
91405 Orsay, France
FILENAME: bbs.neur4.crepel
KEYWORDS: rat, slice, synaptic-plasticity, cerebellum,
excitatory amino-acid receptors, protein kinases, nitric oxide,
cGMP, desensitization.
ABSTRACT: Summary Long-term depression (LTD) of synaptic
transmission at parallel fibre-Purkinje cell synapses is
thought to be a cellular substrate of motor learning in the
cerebellum. This use-dependent change in synaptic efficacy is
induced by conjunctive stimulation of parallel fibres and
climbing fibres. Researchers agree that the induction of LTD
requires, as a initial step, a calcium influx via voltage-gated
Ca 2+ channels into Purkinje cell, together with activation of
ionotropic (AMPA) and probably metabotropic subtypes of
glutamate receptors of this cell. Indeed, due to the lack of
specific antagonist, the final demonstration of the
contribution of metabotropic receptors in the LTD induction
process, under founctional conditions, remains unanswered. The
debate is now focused on the second-messenger processes leading
to LTD of synaptic transmission at parallel fibre-Purkinje cell
synapses, after the calcium influx into the cell. All
researchers agree that a calcium-dependent cascade of events,
including activation of protein kinase C, is necessary for LTD
induction. However, the recruitment in the LTD induction, of
another cascade also triggered by Ca 2+ through synthesis of
nitric oxide and cyclic GMP, remains controversial. On the
other hand, growing evidences suggest that these chains of
reaction underlying LTD migh ultimately lead to a genuine
change in the functional characteristics of AMPA recepotors at
the parallel fibre-Purkinje cell synapses.
============================================================================
(2) MODELS OF THE CEREBELLUM AND MOTOR LEARNING
James C. Houk, Jay T. Buckingham and Andrew G. Barto
Department of Physiology
Northwestern University Medical School
Chicago, Illinois 60611
houk(a)casbah.acns.nwu.edu
Department of Computer Science
University of Massachusetts
Amherst, Massachusetts 01003
buckingham(a)cs.umass.edu
Barto(a)cs.umass.edu
FILENAME: bbs.neur4.houk
ABSTRACT: This article reviews models of the cerebellum and
motor learning starting with the landmark papers by Marr and
Albus and going through present times. The unique architecture
of the cerebellar cortex is ideally suited for pattern
recognition, but how is pattern recognition incorporated into
motor control and learning systems? The present analysis
begins with a discussion of what the cerebellar cortex needs to
regulate through its anatomically defined projections to
premotor networks. Next we examine various models of how the
microcircuitry in the cerebellar cortex may be used to achieve
its regulatory functions. Having thus defined what it is that
Purkinje cells in the cerebellar cortex must learn, we then
evaluate theories of motor learning. We examine current models
of synaptic plasticity, credit assignment and the generation of
training information, indicating how they could function
cooperatively to guide the processes of motor learning.
============================================================================
(3) LONG-LASTING POTENTIATION OF GABAERGIC INHIBITORY
SYNAPTIC TRANSMISSION IN CEREBELLAR PURKINJE CELLS:
ITS PROPERTIES AND POSSIBLE MECHANISMS
Masanobu Kano Department of Physiology
Jichi Medical School
Minamikawachi-machi
Tochigi 329-04, Japan
H00851(a)sinet.ad.jp
FILENAME: bbs.neur4.kano
KEYWORDS: cerebellum, Purkinje cell, inhibitory synapse, GABAA
receptor, long-lasting potentiation, Ca2+, protein kinase
ABSTRACT: The cellular basis of motor learning in the
cerebellum has been attributed mostly to long-term depression
(LTD) at excitatory parallel fiber (PF)-Purkinje cell (PC)
synapses. LTD is induced when PFs are activated in conjunction
with a climbing fiber (CF), the other excitatory input to PCs.
Recently, by using whole- cell patch-clamp recording from PCs
in cerebellar slices, a new form of synaptic plasticity was
discovered. Stimulation of excitatory CFs induced a
long-lasting (usually longer than 30 min) 'rebound
potentiation (RP)' of g-amino-butyric acid A (GABAA)-receptor
mediated inhibitory postsynaptic currents (IPSCs). Similar to
LTD, induction of RP requires transient elevation of
intracellular calcium concentration ([Ca2+]i) due to activation
of voltage-gated Ca2+ channels. Besides, activity of inhibitory
synapses seems to be necessary for RP to occur. RP is mainly
due to up-regulation of postsynaptic GABAA receptor function,
since PC response to bath-applied exogenous GABA is also
potentiated with a time course similar to RP. The difference
in the time scale between the Ca2+ transients (10-30 sec) and
the durations of RP (>30 min) strongly suggests that some
intracellular biochemical machinery is involved. Besides the
well-described LTD, RP could be a cellular mechanism that plays
an important role in motor learning.
============================================================================
(4) CEREBELLAR LONG-TERM DEPRESSION AS INVESTIGATED IN A
CELL CULTURE PREPARATION
David J. Linden
Department of Neuroscience
Johns Hopkins University School of Medicine
725 North Wolfe Street
Baltimore, MD 21205, USA
david.linden(a)qmail.bs.jhu.edu
FILENAME: bbs.neur4.linden
KEYWORDS: Purkinje Neuron, Glutamate, Motor Learning, Synaptic
Plasticity, Climbing Fiber, Parallel Fiber.
ABSTRACT: Cerebellar long-term depression (LTD) is a form of
synaptic plasticity, first described by Ito and co-workers, in
which simultaneous activation of two excitatory inputs to a
Purkinje neuron, the parallel fibers (PF) and the climbing
fibers (CF), results in a sustained depression of PF synaptic
drive. The purpose of this target article is not to assess the
possible role of this synaptic alteration in motor learning, an
issue which is addressed by other authors in this volume, nor
is it to provide a detailed summary of the work on cerebellar
LTD to this point (see Linden and Connor, 1993; Crepel et al.,
1993 for review) or to place cerebellar LTD within the context
of other forms of persistent synaptic depression that occur
within the mammalian brain (see Linden, 1994b). Rather, it is
to discuss results obtained using a very reduced preparation
for the study of LTD, embryonic Purkinje neurons grown in
culture and stimulated with exogenous excitatory amino acids,
and to consider some advantages and limitations of this
approach. Recent work using this preparation has suggested
that three processes are necessary for the induction of
cerebellar LTD, Ca influx through voltage-gated channels, Na
influx through AMPA receptor-associated channels or voltage-
gated Na channels, and protein kinase C activation which is
dependent upon activation of the metabotropic glutamate
receptor mGLUR1. In addition, input-specific induction of LTD
has been demonstrated in this preparation under conditions
where both spontaneous and evoked neurotransmitter release are
reduced or eliminated, indicating that postsynaptic alterations
are sufficient to confer this important computational
property.
============================================================================
(5) ON CLIMBING FIBER SIGNALS AND THEIR CONSEQUENCE(S)
J.I. Simpson, D.R. Wylie, and C.I. De Zeeuw
Dept. of Physiology and Neuroscience
New York University Medical Center
550 First Avenue
New York, NY 10016
simpsj01(a)popmail.med.nyu.edu
Dept. of Psychology,
University of Alberta,
Edmonton, Alberta,
Canada, T6G 2E1.
Dept. of Anatomy,
Erasmus University Rotterdam,
3000 DR Rotterdam,
Postbus 1738,
The Netherlands.
FILENAME: bbs.neur4.simpson
KEYWORDS: cerebellum, Purkinje cells, mossy fibers, synchrony,
complex spikes, simple spikes, movement, posture, inferior
olive, eye movements, flocculus, nodulus
ABSTRACT: The persistence of many contrasting notions of
climbing fiber function after years of investigation testifies
that the issue of climbing fiber contributions to cerebellar
transactions is still unresolved. The proposed capabilities of
the climbing fibers cover an impressive spectrum. For many
researchers, the climbing fibers signal errors in motor
performance, either in the conventional manner of frequency
modulation or as a single announcement of an "unexpected
event". More controversial is the effect of these signals on
the simple spike modulation of Purkinje cells. In some hands,
they lead to a long-term depression of the strength of parallel
fiber synapses, while in other hands they lead to a
short-lasting enhancement of the responsiveness of Purkinje
cells to mossy fiber inputs, or contribute to the often seen
reciprocal relation between complex and simple spike
modulation. For yet other investigators, the climbing fibers
serve internal timing functions through their capacity for
synchronous and rhythmic firing. The above viewpoints are
presented in the spirit of trying to reach some consensus about
climbing fiber function. Each point of view is introduced by
summarizing the key observations made by the respective
proponents and then the issues of short-lasting enhancement,
reciprocity between complex and simple spikes, and synchrony
and rhythmicity are addressed in the context of the visual
climbing fiber system of the vestibulocerebellum.
============================================================================
(6) DOES THE CEREBELLUM LEARN STRATEGIES FOR THE OPTIMAL TIME-VARYING
CONTROL OF JOINT STIFFNESS?
Allan M. Smith
Centre de recherche en sciences neurologiques
Departement de physiologie
Universite de Montreal
C.P. 6128, Succ. A
Montreal (Quebec)
H3C 3J7
smitha(a)ere.umontreal.ca
FILENAME: bbs.neur4.smith
KEYWORDS: muscle synergies, agonist-antagonist co-contraction,
joint stiffness, muscle activation strategies, multjoint limb
control, cerebellum, motor learning
ABSTRACT: Although there is increasing agreement that the
cerebellum plays an important role in motor learning, the
basic substance of what constitutes motor learning has been
difficult to define. Unless motor learning is somehow
radically different from other forms of learning, it must
involve relatively simple stimulus-stimulus and
stimulus-response associations. All forms of learning,
including purely sensory associations and cognitive learning
as well as motor learning effect changes in behavior
generally. However, a singular characteristic of motor
learning is that it adjusts joint and limb mechanics by
altering the neural input to muscles through practice and
mental rehearsal. The hypothesis proposed here is that the
cerebellum plays an important role in motor learning by
forming and storing associated muscle activation patterns for
the time-varying control of limb mechanics. By modulating the
co-contraction of agonist-antagonist muscles through
adjustments in the timing and amplitude of muscle activity, the
viscoelastic properties of joints can be appropriately
regulated throughout movement and adapted for transitions
between postures and movements. Optimal control of joint
viscoelastic properties cannot be totally achieved by on-line
corrections initiated by reflex feedback, because of the delays
and consequent instabilities incurred. Instead, strategies for
optimizing muscle activation patterns or synergies must be
learned from the temporal association of proprioceptive stimuli
signaling muscle lengths and forces and the rates of changes in
these parameters with the reinforcement occurring when the
movement achieves its objective. Such strategies would involve
varying degrees of co-contraction or reciprocal inhibition of
agonist-antagonist muscles which ultimately contribute to the
joint and limb stiffness. Evidence from neural recordings and
clinical and experimental lesion studies are presented
suggesting that the cerebellum uses teleceptive and
proprioceptive feedback as feedforward conditioned stimuli for
specific muscle activation patterns contributing to joint
stiffness (i.e. agonist-antagonist muscle synergies) for
particular tasks and postures. A wide variety of observations
are thought to be consistent with such a role for the
cerebellum, but ultimately additional experiments could prove
or disprove this hypothesis.
============================================================================
(7) ON THE SPECIFIC ROLE OF THE CEREBELLUM IN MOTOR LEARNING AND
COGNITION: CLUES FROM PET ACTIVATION AND LESION STUDIES IN MAN
W. T. Thach
Department of Anatomy and Neurobiology
Washington University School of Medicine
660 South Euclid Avenue
St. Louis, MO 63110
thachw(a)thalamus.wustl.edu
FILENAME: bbs.neur4.thach
KEYWORDS: cerebellum, motor learning, mental movement imagery,
planning, sequence, timing, cognition.
ABSTRACT: Brindley proposed that we initially generate
movements "consciously", under higher cerebral control. As the
movement is practiced, the cerebellum learns to link within
itself the context in which the movement is made to the lower
level movement generators. Marr and Albus proposed that the
linkage is established by a special input from the inferior
olive which plays upon an input-output element within the
cerebellum during the period of the learning. When the linkage
is complete, the occurrence of the context (represented by a
certain input to the cerebellum) will trigger (through the
cerebellum) the appropriate motor response. The "learned"
movement is distinguished from the "unlearned" conscious
movement by its now being automatic, rapid, and stereotyped.
The idea is still controversial, but has been supported by a
variety of animal studies and, as reviewed here, is consistent
with the results of a number of human PET and ablation
studies. We add to the idea of context-response linkage what
we think is another important variable: novel combinations of
downstream elements. With regard to the motor system and the
muscles, this could explain how varied combinations of muscles
may become active in precise time-amplitude specifications so
as to produce coordinated movements appropriate to specific
contexts. In this article we further extend this idea to the
premotor parts of the brain, and their role in cognition.
These areas receive influences from the cerebellum, are active
both in planning movements that are to be executed, and in
thinking about movements that are not to be executed. From
recent evidence, the cerebellar output extends even to what has
been characterized as the ultimate frontal planning area, the
"prefrontal" cortex, area 46. The cerebellum thus may be
involved in context-response linkage, and response combination
even at these higher levels. The implication would be that,
through practice, an experiential context would automatically
evoke a certain mental action plan. The plan would be in the
realm of thought, and either could--or need not--lead to
execution. The specific cerebellar contribution would be one
of the context linkage and the shaping of the response, through
trial and error learning. The prefrontal and premotor areas
could still plan without the help of the cerebellum, but not so
automatically, rapidly, stereotypically, so precisely linked to
context, or so free of error. Nor would their activities
improve optimally with mental practice.
============================================================================
(8) NITRIC OXIDE AND SYNAPTIC PLASTICITY; NO NEWS FROM THE CEREBELLUM
Steven R. Vincent
Division of Neurological Sciences
Department of Psychiatry
University of British Columbia
2255 Wesbrook Mall
Vancouver, B.C. V6T 1Z3 Canada
steven.r.vincent(a)mtsg.ubc.ca
FILENAME: bbs.neur4.vincent
KEYWORDS: Nitric oxide, cyclic guanosine monophosphate (cGMP),
cerebellar cortex, synaptic plasticity, guanylyl cyclase
ABSTRACT: Interest in the role of nitric oxide (NO) in the
nervous system began with the demonstration that glutamate
receptor activation in cerebellar slices causes the formation
of a diffusible messenger with properties similar to those of
the endothelial derived relaxing factor. It is now clear that
this is due to the Ca2+/calmodulin-dependent activation of the
enzyme NO synthase, which forms NO and citrulline from the
amino acid L-arginine. The cerebellum has very high levels of
NO synthase, and although it has low levels of guanylyl
cyclase, cerebellar cGMP levels are an order of magnitude
higher than in other brain regions. A transcellular metabolic
pathway is also present in the cerebellar cortex to recycle
citrulline back to arginine. The NO formed binds to and
activates soluble guanylyl cyclase to elevate cGMP levels in
target cells. Studies employing NADPH-diaphorase, a selective
histochemical marker for NO synthase, together with
immunohistochemistry, in situ hybridization and biochemical
studies have indicated that NO production occurs in granule and
basket cells in the cerebellar cortex, while cGMP formation
appears to occur largely in other cells, including Purkinje
cells. Given that a long-term depression of AMPA currents can
be seen in isolated Purkinje cells, this anatomical
localization suggests that NO cannot play an essential role in
the induction of this form of synaptic plasticity.
============================================================================
============================================================================
To help you decide whether you would be an appropriate commentator for
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ftp.princeton.edu according to the instructions below. The filenames are:
bbs.neur4.crepel
bbs.neur4.houk
bbs.neur4.kano
bbs.neur4.linden
bbs.neur4.simpson
bbs.neur4.smith
bbs.neur4.thach
bbs.neur4.vincent
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you feel you would bring to bear on what aspect of which article(s).
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-------------------------------------------------------------
From: KUNLT1::PBODA 8-AUG-1995 19:54:23.44
To: PBODA
CC:
Subj: 1
Egy holland srac szeretne szakmai gyakorlatat Budapesten tolteni,
a szakmai gyakorlatnak bizonyos fokig onallo kutatast/munkat is
magaba kell foglalnia.
Terulete: 'cognitive science',
amiket azon belul tanult: 'psychology, AI, psycholinguistics, neural networks'
amit gyakorlatilag szeretne tenni: 'human-computer interaction
with graphical interfaces'.
Kerdese a kovetkezo: van-e olyan intezmeny Budapesten amelyik fogadna ot
kb. 6 honapra. Anyagiag: ha kapna valamilyen nevleges kis osztondijat,
az csak jol jon, egyebkent nem tart ra igenyt.
Ha barkinek volna otlete/ismerose, kerem irjon.
Boda Peter
Nijmegen, Hollandia